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Well, this is getting really serious: Coronavirus (now increasingly known as COVID-19) has reached Washington, DC [Rector of prominent Washington, D.C., church tests positive for coronavirus, CBS, March 8, 2020; CPAC chair had brief contact with coronavirus patient at conference, says he’s not alarmed, Fox News, March 8, 2020]. Global panic is in the air: Italy, which appears to be peculiarly vulnerable to COVID-19 as it was to the 1918 Spanish Flu Pandemic , has reacted with arguably counter-productive ferocity [Leaked coronavirus plan to quarantine 16m sparks chaos in Italy, Guardian, March 8, 2020]. But, although totally repressed by our race-denying Ruling Class—did you know that Kirkland WA, where the bulk of U.S. deaths have occurred, was 11.3% Asian in the 2010 census?—the evidence that there may be an ethnic and/or racial dimension to the disease continues to mount. Which could mean that the Ruling Class is frightening most people too much—and not warning some people enough.
The evidence has been helpfully brought together by Canadian blogger and independent researcher Peter Frost, who has published in such scientific journals as the Journal of Circumpolar Health, on his blog Evo and Proud [Coevolution with the plague, by Peter Frost, Evo and Proud, March 2, 2020].
Frost reports that there have now been three studies which have attempted to test whether or not there might be genetically-based race differences in susceptibility to COVID-19. The first is the one which I discussed last week, [Single-cell RNA expression profiling of ACE2, the putative receptor of Wuhan 2019-nCov, by Yu Zhao et al., bioRxiv, 2020]. As Frost noted in a comment on my piece at the Unz Review:
The authors found that the ACE2 receptor is concentrated in certain cells and that the number of such cells in lung tissue was five times greater in the Asian donor. There was only one Asian donor in the entire sample, and no further description is provided on this “Asian.” However, the chances are low that the same normal distribution would produce such an extreme outlier.
So, we can be cautiously confident that this study provides of evidence for there being race differences in susceptibility to the virus.
A second study, Frost observes, appeared to refute the first. It did not find “significant differences in ACE2 receptor gene expression between Asian and Caucasian lung tissue.” [Bulk and single-cell transcriptomics identify tobacco-use disparity in lung gene expression of ACE2, the receptor of 2019-nCov, by G. Cai, medRxiv, 2020].
However, explains Frost, the problem is that the sample employed from the US and, thus, the term “Asian” may have conflated “East Asian” and “South Asian,” despite these being distinct races. This is a problem I have looked at before.
The third study, which actually has been peer-reviewed and which employed different methods from the first, found that there were large differences between Asians and Non-Asians, and also within the Asian category, with regard to the distribution of the ACE2-receptor [Comparative genetic analysis of the novel coronavirus (2019-nCoV/SARS-CoV-2) receptor ACE2 in different populations, by Y. Cao et al., Cell Discovery, 2020].
Specifically, the authors found that the prevalence of these receptors was greatest among Han Chinese, then “Mixed American” (U.S. population, not broken-up by race) then African, and finally European. The difference between the Chinese and Non-Chinese samples was substantial.
So we have yet more evidence that COVID-19 discriminates on the basis of race. And, in that races are genetic clusters evolved to different ecologies, it probably also does so with regard to the less stark genetic clusters that constitute ethnic groups within races. Contemporary observers actually noted this with regard to the impact of the 1918 Spanish flu on poor immigrants to the US from assorted ethnic backgrounds, when Italian-Americans were particularly vulnerable.
On Evo and Proud, Frost presents a novel argument regarding why COVID-19 has been so devastating in China. He argues that “coronaviruses,” such as the common cold and flu, have “coevolved with the Chinese population.” Repeated exposure of the lung tissue to these viruses builds up resistance to them and, by extension, increases resistance to more dangerous novel versions of them as well as to other “serious pulmonary infections” such as TB.
In other words, the Chinese are evolved to easily become infected by coronaviruses, such that resistance to more virulent pulmonary conditions can be accrued. This has been their evolutionary strategy.
It follows, argues Frost, that modern medicine, by reducing the prevalence and severity of milder viral infections, has left the Chinese—though not necessarily groups who have evolved a different strategy – with insufficient resistance to COVID-19.
Frost shows that this process, of one virus making you more immune to a subsequent different but related virus, has been seen in mice. Evidence for his thesis can also be seen in the modern Chinese being especially impacted by many other flu-like viruses.
Frost speculates, based on this, that:
“In particular, crowded environments, with high rates of life-threatening pulmonary infections, notably tuberculosis, pneumonia, and pneumonic plague, should have favored individuals who are more susceptible to infection by coronaviruses. Historically, such environments would encompass not only China but also other areas that have long had large urban populations and a correspondingly long coevolution with pulmonary infections. These areas would notably include the Indo-Gangetic Plain in India and the Fertile Crescent of the Middle East.” [Coevolution with the plague, March 2, 2020].
Perhaps even Italy, relatively close to the Fertile Crescent from which civilization spread, might be in this category.
Northern Europeans, of course, urbanized rather later and adapted to crowded environments rather later. Accordingly, it seems that they did not evolve this propensity to easily catch coronaviruses. With flu being by prevalent in such ecologies due to the weather, and plagues not being such a problem due to sparser populations, it may be that they evolved to avoid catching the flu, specifically.
Moreover, being strongly exposed to flu, Northern Europeans may have evolved to be strongly resistant to it if and when they caught it at all.
It would follow that the arrival of modern medicine would present a serious flu hazard for people closer to the Fertile Crescent—because they would be evolved to catch flu as a means of rendering themselves resistant to forms of pestilence associated with large populations—but that it would not be such an issue further north (or in the Tropics) where people would simply be evolved to not catch flu.
Frost has certainly presented an intriguing hypothesis and our Ruling Class is certainly not going to talk about it.
It would imply that, although members of all races and ethnic groups may die of COVID-19, certain races and ethnic groups might be significantly worse impacted for genetic reasons.
In other words, it would imply that race is real. And we can’t have that.
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This is a tautological pronouncement. Not a testable hypothesis unless you also hypothesize why other populations did not “coevolve” in similar way. At this point like pretty much everything that Frost produces this is no different than Kipling’s just-so stories. But nincompoops like Lance Welton will eat up any just-so story that confirms their bias.
1) Hu – Lance Welton, I have to admit, now you have allies. – You put me on a rollercoaster ride with the pros and cons of virus susceptibility along racially defined – hehe – borders. At times I almost thought, your hypothesis does not hold up.
2) That’d be the second aspect, the HBD-deniers would have a hard time to admit: That real-world borders still matter.
First German died in Egypt yesterday. However, he was probably a boomer, and boomers aren’t really white.
And of course neither are Italians, Anglos, or Amerimutts.
Chad Welton still going strong!
Well, he did explain with the example of Nordic populations.
This is the worst written article that I have read here, e.g.,
In other words, the Chinese are evolved to easily become infected by coronaviruses, such that resistance to more virulent pulmonary conditions can be accrued. This has been their evolutionary strategy.
The us regime (who has a long sordid history of genocide using biological warfare) is currently trying to crush Iran and China to maintain full spectrum imperial tyranny. During this time, both those targets of the us regime are hit by a virulent coronavirus that “coincidentally” has a high lethality rate against ethnic Han Chinese and Iranians (and Italians who share very similar genetics with Iranians).
This coronavirus also “coincidentally” mostly skips over Europeans, Israelis, and Africans.
Sure, it’s all just a “coincidence” along with the simultaneous viruses that decimated China’s pig farms and chicken farms. Now, China needs to buy pigs and chickens from us farms. This all happened “coincidentally” during the us regime’s trade war.
Don’t worry everyone. The us would never behave like a blood thirsty psychopath to over throw foreign nations.
It’s all just a “coincidence”. Nothing to see here.
Anyone find those wmd in Iraq yet?
Italy and Iran have more cases than most East Asian countries and possibly a higher death ratio. The evidence so far suggests Caucasoids are as vulnerable to the virus as East Asians if not more so.
Exactly what I have been saying. The root of our civilisation is a white healthy man called Adam with a ribbon called Eve.
Then Eve sleep with another monkey behind Adams back, and we have all the trouble with Cro-Magnon races, Feminism and Spaghetti everywhere.
This seems to be what the white guys in London and Washington wants to rectify.
The problem with this theory is that China historically has been very rural. As recently as the 1980s, China was 80%~90% rural.
Whereas certain parts of Northern Europe such as England were significantly urbanized by the early modern era and Industrial Revolution.
During the Spanish Flu, China was around 95% rural, while the US was around 40%~50% urbanized. America was an urbanized, industrial behemoth with major metropolises, motor vehicle transport, and transcontinental railroads. China was a country of rural peasants. That would explain the difference in the spread and severity of the Spanish Flu between the US and China.
China’s urbanization has taken place rapidly over the past 30 years, while urbanization in the US and certain areas of Northern Europe like England has occurred for centuries by now.
Another explanation is that it was less thoroughly recorded in backwards, rural China.
It’s sometimes asserted that the Spanish Flu was less severe in Japan either. But recently I read that some researchers have started to question that. There were simply much worse statistics than in Europe. The same could be even more true of China.
Lance Welton is the most based and redpilled person I’ve ever seen anywhere.
Fortunately there are some other based and redpilled takes on Corona-Chan:
Interestingly, I see lots of liberals spouting the “iT’S jUsT THe fLU” opinion, especially the I Fucking Love Science crowd, the very rational atheists, who will rationally count the number of deaths from terrorism or Covid-19, and then proclaim that more people drown in the home pools, so we should be more afraid of pools than terrorism or Covid-19, because it’s “irrational.”
So Trump is basically making a liberal talking point. He could’ve made it a talking point about the dangers of globalism, immigration, global supply chains, etc., but he sadly chose to use the stock market as the only metric of his own performance, and will now be probably judged on that.
Since 80-85% of the people who are infected just get the sniffles, the genetic issue is what population is susceptible to cytokine storms. Cytokine storms are what kill and overwhelm healthcare systems.
https://www.uab.edu/reporter/know-more/publications/item/8909-here-s-a-playbook-for-stopping-deadly-cytokine-storm-syndrome
Also:
“One in seven” is in the range of 10-15%.
https://www.zerohedge.com/geopolitical/scientists-warn-about-dangerous-tipping-point-where-covid-19-goes-mild-deadly
“Italy, which appears to be peculiarly vulnerable to COVID-19 as it was to the 1918 Spanish Flu Pandemic” Italy has 250,000 Chinese people that live in milan, the government failed to properly follow quarantine procedures. Also Italy has a higher population density than most European countries which would facilitate the spread of the virus. Also during the Spanish Flu pandemic European countries like Germany, France and England suffered catastrophically. Infecting over 800,000 people in Ireland in a two month period. Killing 125,000 to 250,000 French civilians and 30,000 French soldiers, claiming the lives of 228,000 British, and more than 400,000 German civilians.
want to suggest the OPPOSITE: that Chinese are less likely to succumb to the virus, but Europeans are MORE likely to fall to the virus, based on previous findings for SARS.
https://www.nature.com/articles/ng1698
Severe acute respiratory syndrome (SARS) is caused by infection of a previously undescribed coronavirus (CoV). L-SIGN, encoded by CLEC4M (also known as CD209L), is a SARS-CoV binding receptor that has polymorphism in its extracellular neck region encoded by the tandem repeat domain in exon 4. Our genetic risk association study shows that individuals homozygous for CLEC4M tandem repeats are less susceptible to SARS infection. L-SIGN is expressed in both non-SARS and SARS-CoV–infected lung. Compared with cells heterozygous for L-SIGN, cells homozygous for L-SIGN show higher binding capacity for SARS-CoV, higher proteasome-dependent viral degradation and a lower capacity for trans infection. Thus, homozygosity for L-SIGN plays a protective role during SARS infection.